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Table 3 Expression of IFI16 was significantly high in HBV-GN compared with CGN

From: IFI16 induces inflammation in hepatitis B virus-associated glomerulonephritis by regulating the Caspase-1/ IL-1 ß pathway

group

Tissue

Cases(n)

Age

Gender (M %)

IFI16

Positive

Rate(%)

+

++

+++

HBV-GN

K

50

36.1 ± 12.7

35 (70%)

10

14

26

0

80

CGN

K

25

38.2 ± 15.5*

18 (72%)**

19

6

0

0

24***

  1. HBV-GN Hepatitis B viral associated glomerulonephritis, CGN Chronic glomerulonephritis, K kidney, M Male. *compared with HBV-GN, (t = −0.072, p = 0.943); **compared with HBV-GN, (×2 = 0.032, p = 0.85); ***compared with HBV-GN, N, (× 2 = 30.091, p < 0.01)